Preliminary experiments show conolidine may possibly inhibit unique ion channels, lowering neuronal excitability and limiting pain signals. This mechanism is particularly suitable in neuropathic pain, where by abnormal signaling will cause persistent pain. On top of that, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to discomfort https://johnn903jjj6.wikiusnews.com/user